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  • Re: The Carbon Dioxide level that initiates Hyperventilation

    "It was found that when the proportion of CO2 in the air rose to about
    3% and the oxygen fell simultaneously to about 17% "... the breathing
    began to be noticeably increased ... until with about 6% CO2 and 13% of
    oxygen the panting was very "great ...

    Page 16 in J S Haldane & J G Priestley "Respiration" New edition.
    (Oxford University Press, 1935)

    Probably from 2 articles in J.Path. Bact. (1893) 1: 168 & 318

    Adrian Smith
    Leeds University Library
    UK

    -----Original Message-----
    From: * Biomechanics and Movement Science listserver
    [mailto:BIOMCH-L@NIC.SURFNET.NL] On Behalf Of McFarlane, David
    Sent: 15 November 2006 00:59
    To: BIOMCH-L@NIC.SURFNET.NL
    Subject: [BIOMCH-L] The Carbon Dioxide level that initiates
    Hyperventilation


    Dear all,

    Does anyone know the level of carbon dioxide in air that initiates
    hyperventilation? I have looked at my old industrial hygiene studies on
    the sick building syndrome I have found that some organizations have
    given recommended maximum figure for CO2.

    According to my records the seminar edition of the publication "Managing
    Indoor Air Quality" (BOMA, 1991) cited the NHMRC (Australian "National
    Health and Medical Research Council) and Worksafe Australia
    recommendations. Worksafe Australia (1990) had recommended a figure of
    5,000 ppm for long-term exposure to CO2 at that time. On the basis of
    anecdotal evidence (from deep sea diving, mining disasters and submarine
    mishaps) that figure looks rather pessimistic (a modest colleague tells
    me that he survived 15, 000 ppm for 15 minutes after a mine disaster
    with no obvious ill effects and the U Boat crews of WW2 supposedly
    endured levels up to nearly 2%; "Das Boot" et cetera).

    Incidentally I often wonder what sort of data these long-term exposure
    thresh-holds were based on (I have not found any recent examples of good
    scientific research papers on this topic yet). Does anyone have any
    idea?

    Are there any work physiologists out there who are still researching
    this topic? Back in the seventies when I was ay uni physiologists had
    supposed that the blood vessels of the lungs contained CO2-sensitive or
    acidosis-sensitive sensors that regulated the rate and depth of
    breathing (rather than a direct response to anoxia). Can anyone tell me
    what science has found in the meanwhile? If not is anyone looking for a
    PhD thesis?


    Regards,
    David McFarlane
    Ergonomist, WorkCover NSW

    References

    1. Building Owners and Managers Association of Australia, (1991),
    "Managing Indoor Air Quality", section 9.3, page 66.

    2. Worksafe Australia Standard, (1990), "Exposure contaminants for
    atmospheric contaminants in the occupational environment".



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